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. 2018 Aug 12;7:146. doi: 10.1038/s41426-018-0146-6

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Major regions of divergence are labeled, including: (1) An 1.5-Kb deletion in the K. pneumoniae 4 genome with two genes encoding hypothetical proteins (indicated with a black arrow); (2) disruption of the mgrB gene by insertion sequence (ISKpn18) in the K. pneumoniae 5 genome (indicated with a black arrow); (3) A 16.5-Kb region of the K. pneumoniae 5 genome (indicated by a blue rectangle) is absent in the other two ST11 K. pneumoniae genomes. Predicted genes within this region are indicated: (a) peptidase; (b) fatty acid desaturase; (c) phosphate ABC transporter substrate-binding protein; (d) nitrilotriacetate monooxygenase; (e) hypothetical protein; (f) transcriptional regulator TdcA; (g) bifunctional threonine ammonia-lyase/L-serine ammonia-lyase TdcB; (h) threonine/serine transporter TdcC; (i) propionate kinase; (j) formate C-acetyltransferase; (k) branched-chain amino acid ABC transporter substrate-binding protein; (l) branched-chain amino acid ABC transporter permease, LivH; (m) branched-chain amino acid ABC transporter permease, LivM; (n) ABC transporter ATP-binding protein, LivG; (o) ABC transporter ATP-binding protein, LivF