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. 2018 Aug 2;15:119. doi: 10.1186/s12985-018-1025-4

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — Acute replication in mice of PRV JS-2012, UL41 revertant virus and UL41 null virus. a Weight changes of infected mice during the period of acute infection. The data were presented as means ± SE for weight changes of 5 random selected mice per data point. Asterisk (*) indicated a significant difference between JS-2012 and UL41 null virus, and pound (#) indicated a significant difference between UL41 revertant and UL41 null viruses. Viral titers in the trigeminal ganglia (b), brain stem (c), Olfactory bulb (d) of mice infected with PRV strain JS-2012 (wild type), UL41 revertant virus or UL41 null virus were shown. The data represented means ± SE for 5 samples per data point. For panel a, the student’s t-test was used for analyzing the data; for panel b, two-way ANOVA was used for analyzing the data, **P < 0.01