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. 2018 Aug 3;10:103. doi: 10.1186/s13148-018-0535-7

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 2 — The methylation status of TET1 in NSD, surgical margin tissues, nasopharyngeal carcinoma (NPC) tissues, and nasopharyngeal carcinoma cells lines. a Promoter methylation of TET1 in surgical margin tissues and nasopharyngeal carcinoma tissues as measured by MSP. M: methylated; U: unmethylated. b Bisulfite genomic sequencing confirmed the methylation status of TET1 CpG sites in NSD, NPC tissues, and HONE1 cells with or without A+T treatment