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. 2018 Aug 14;18:117. doi: 10.1186/s12935-018-0615-y

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© The Author(s) 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — The JNK/F-actin pathway is also involved in mitochondrial homeostasis. a, b JC-1 staining for mitochondrial membrane potential. Red fluorescence indicates normal mitochondria and green fluorescence indicates damaged mitochondria. c, d ROS production was evaluated via immunofluorescence. e Caspase-9 activity was measured to assess mitochondrial apoptosis. Inhibition of the JNK pathway abrogated the pro-apoptotic effects of TAZ on mitochondria in HepG2 cell. *P < 0.05 vs. control (Ctrl) group; ^#P < 0.05 vs. IL-2 group; ^@P < 0.05 vs. IL-2 + si-TAZ group