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Post-reperfusion DEX treatment attenuated the increase in CTP1A, NOX4, and TXNIP in renal IR injury (A) and reversed the increase in phospho-AKT and phospho-ERK levels in renal IR injury (B). *p < 0.05, compared with D-sham baseline; †p < 0.05, compared with D-IRC group; ‡p < 0.05, compared with DEX pre-treatment group.
