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. 2018 Aug 16;8:12252. doi: 10.1038/s41598-018-29708-9

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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TN tumors expressing low levels of miR-342-3p are enriched in glycolysis pathway. (a) Z-score values of single sample GSEA analysis (ssGSEA) based on Glycolysis terms of TN tumors from our cohort, METABRIC and TCGA database stratified by miR-342-3p expression: low (≤1st quantile), intermediate (>1st quantile but <3^rd quantile) and high (≥3^rd quantile). (b) Correlation between miR-342-3p, MCT1 and glycolysis ssGSEA score (c) Chord plot illustrating the significant enriched pathways terms that are overrepresented in tumors with low miR-342-3p level (blue) and tumors with high miR-342-3p level (red) (tumors from our cohort, TCGA and METABRIC databases) (d) General conclusion of the descriptive genomics of TN tumors in accordance to miR-342-3p expression. Heatmap of the expression of miR-342-3p and its target gene MCT1 in profiled tumors (our cohort, METABRIC and TCGA) divided by miR-342- 3p expression level. The relative abundances of miR-342-3p and MCT1 are inversely based on miR-342-3p expression level.