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. 2018 Jun 19;16(3):2903–2910. doi: 10.3892/ol.2018.8989

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — SMOC1 silencing reduces the ROS content in Ang II-induced MFBs. Flow cytometry was performed on the ROS content in MFBs, MFBs transfected with an empty vector, and Ang II-induced MFBs transfected with an empty vector or si-SMOC1. **P<0.01 and ***P<0.001 vs. NC; ^{^^}P<0.01 vs. Ang II+NC. SMOC1, SPARC-related modular calcium binding 1; ROS, reactive oxygen species; MFBs, myocardial fibroblasts; si, small interfering RNA; Ang II, angiotensin II; NC, negative control.