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. 2018 Aug 14;14:1744806918783478. doi: 10.1177/1744806918783478

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© The Author(s) 2018

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — Chronic inflammation model mice reduced the frequency of mIPSCs in the ACC. (A) Recorded mIPSCs holding potential at 0 mV in a layer II/III pyramidal neuron from control (a) or CFA model in the presence of TTX (1 μM) (b). (B) The cumulative probability in inter-event intervals between control and CFA group (a). CFA group reduced the average frequency of mIPSCs compared with control group (b). The cumulative probability in amplitude between control and CFA group (c). There was no difference in average amplitudes of mIPSCs between control and CFA group (d). Results are expressed as the mean ± SEM. Asterisks shows P< 0.05.

mIPSCs: miniature inhibitory postsynaptic currents; CFA: complete Freund adjuvant; NS: not significant.