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Proposed model of action of CNPs as redox-independent radio-sensitizing agents in HaCat keratinocytes cells. CNPs administration may promote in a redox-independent fashion the strengthening of cell DNA damage response (DDR) after exposure to radiations, diminishing X-ray-induced DNA lesions on one side, and increasing the stringency of cell cycle checkpoints and forcing damaged cells to undergo apoptosis on the other, thus preventing radiation-induced mutagenesis.
