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. 2018 Aug 21;8:12523. doi: 10.1038/s41598-018-30413-w

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Schematic representation of the putative resistance mechanism of Mgaloblishvili (in green) against P. viticola (in purple) based on the overexpression of genes involved in the plant defense pathway at 1 dai. (1) Pathogen recognition through PAMP, DAMP and effector receptors, and ubiquitination. (2) Phytohormone signaling based on ethylene. (3) Resistance response based on synthesis of antimicrobial compounds and fungal wall degradating enzymes, and cell wall reinforcement. (4) Systemic signaling based on the indirect activation of the disease resistance protein PR1 that is involved in Systemic Acquired Resistance (SAR).