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. 2018 Feb 3;9(2):551–564. doi: 10.1007/s13300-018-0370-z

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© The Author(s) 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (https://creativecommons.org/licenses/by/4.0), which permits use, duplication, adaptation, distribution, and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 1 — a, b Mean (± SE) HR over time: analysis by GLP-1RAs vs. non-GLP-1RA treatments (a) and by individual GLP-1RAs vs. non-GLP-1RA treatments (b). c Box and whisker plot of change in HR from baseline at each time point. d, e LS mean (± SE) change in HR from baseline to final on-treatment HR measurement by GLP-1RAs vs. non-GLP-1RA treatments (d) and by individual GLP-1RAs vs. non-GLP-1RA treatments (e). f, g LS mean (± SE) change in HR over time in studies with posttreatment follow-up for GLP-1RA vs. non-GLP-1RA treatments (f) and individual GLP-1RAs vs. non-GLP-1RA treatments (g). *P < 0.0001 vs. non-GLP-1RA treatments. BID twice daily, BL baseline bpm beats per minute, EOT end of treatment, GLP-1RA glucagon-like peptide-1 receptor agonist, HR heart rate, LS least-squares, QW once weekly, SE standard error