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. 2018 Aug 22;8:12564. doi: 10.1038/s41598-018-30021-8

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Senofilcon A UVB blocking contact lenses protect against changes in the metabolic activity and rate of scratch wound closure of limbal epithelial cells and fibroblasts. (A,B) Alamar Blue data showed that the metabolic activity of HLE (A) and HLF (B) cells which were protected by the UVBCL contact lens was maintained in similar levels to the BT and NO UV controls (20 mJ/cm²). (C,D) HLE cells exhibited non-significant changes in wound healing activity at neither 4 h nor 8 h time-point following UVB irradiation (C). HLF cells protected by CL contact lenses featured slower wound closure compared to cells covered by UVBCL lenses, BT and cells that were not UV treated (NO UV) (D). n = 6, *p < 0.05, **p < 0.01, ***p < 0.001, ****p < 0.0001.