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. 2018 Aug 24;8:12738. doi: 10.1038/s41598-018-31121-1

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Agonistic cMet Ab and rHGF protects human glomerular endothelial cells (GECs) against fibrosis in an in vitro model. (A–D) After fibrosis induction with TGF-β in GECs, agonistic cMet Ab (A,C) and rHGF (B,D) ameliorated the fibrosis. The cropped blots are used in the figure, and full-length blots are presented in Supplementary Figs S2–S3, respectively. All values are presented as the means ± SEM (n = 4 per group for each experiment). *P < 0.05, **P < 0.01.