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Erlotinib effectively inhibited EGFR tyrosine kinase activity in eNOS−/−db/db mice. A: Erlotinib inhibited the levels of phosphorylated EGFR (p-EGFR) at different tyrosine residues. B: Erlotinib treatment led to decreased levels of phosphorylated extracellular signal–regulated kinase (p-ERK), a downstream signaling target of EGFR activation.
