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. 2018 Jul 23;40(4):2023–2035. doi: 10.3892/or.2018.6590

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Copyright: © Liu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — CLEC3B copy number variation analysis using TCGA datasets. (A) Deep deletion of the copy number for CLEC3B and VHL in patients with ccRCC from the a TCGA kidney renal clear cell carcinoma dataset. (B) A significant correlation between the residual copy numbers of CLEC3B and VHL was identified using Pearson's correlation analysis. (C) The frequency of deep deletion in the CLEC3B copy number was substantially higher in ccRCC compared with other cancer types. (D) CLEC3B copy number in ccRCC but not pRCC was signifcantly lower compared with normal tissues. CLEC3B, C-Type Lectin Domain Family 3 Member B; VHL, von Hippel-Lindau Tumor Suppressor; TCGA, The Cancer Genome Atlas; ccRCC, clear cell renal cell carcinoma; pRCC, papillary renal cell carcinoma.