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. 2018 Jul 23;40(4):2023–2035. doi: 10.3892/or.2018.6590

Figure 4.

Figure 4.

CLEC3B expression inhibits ccRCC cell proliferation in vitro. (A) Endogenous CLEC3B expression status in five ccRCC cell lines. (B) Western blot analysis revealed the ectopic expression of exogenous CLEC3B in CLEC3B-transfected 786-O and 769-P cells. (C) Effect of CLEC3B overexpression on the growth of 786-O and 769-P cells as measured by a real-time cell analyzer system. (D) Expression of Ki-67, p38, p-p38, ERK and p-ERK, was detected by western blot analysis in CLEC3B-transfected 786-O cells. (E) Semi-quantitative analysis of western blot analysis. *P<0.05 and **P<0.01 with comparisons shown by lines. CLEC3B, C-Type Lectin Domain Family 3 Member B; NC, negative control; p-, phosphorylated; ERK, extracellular signal-regulated kinases; ccRCC, clear cell renal cell carcinoma.