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. 2018 Aug 28;9(9):851. doi: 10.1038/s41419-018-0937-7

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Tumor nodes with DSCR8 clones had larger tumor volume (middle panel) and heavier weight (right panel) than these with vectors. b Tumor nodes with shVector-Huh7 had larger tumor volume (middle panel) and heavier weight (right panel) than these with sh-DSCR8. Immunohistochemical staining in the xenografted tissues for Ki-67 (magnification: ×400. Bars: 50 µM.) in tumor nodule tissues showed that DSCR8 clone increased (c), whereas sh-DSCR8 decreased the percentage of Ki-67-positive cells (d). TUNEL assay in the xenografted tissues (magnification: ×400. Bars: 50 µM.) showed that DSCR8 clone reduced the proportion of apoptotic cells (e), whereas sh-DSCR8 increased the percentage (f). Bars: 50 µM. *P < 0.05, **P < 0.01, ***P < 0.001