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. 2018 Sep 3;8:13159. doi: 10.1038/s41598-018-31620-1

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Myb-inhibitory activity of selected compounds. The figure shows the chemical structures of toyocamycin and teniposide (a), the luciferase activity of Hek-Myb-Luc cells (b) or Hek-Luc cells (c) after treatment for 12 hours with the respective compound and the impact of each compound on the viability of the Hek-Myb-Luc cells (d). The bottom panels in b show the expression of MYB and β-actin. Columns and standard deviations for luciferase activity are based on three independent experiments with two replicate samples in each case. Viability assays are based on two independent experiments with six replicate samples in each case.