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. 2018 Jul 10;1:87. doi: 10.1038/s42003-018-0094-7

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Hypothetical model for the evolution of unique rDNAs/rRNAs in AMF. Evolutionary model for the lack of TRSs in AMF and its sequence heterogeneity. The various environmental conditions (e.g., various host species) may lead to the evolution of phenotypic plasticity via multiple types of ribosomes in AMF. If the rDNA is exposed to disruptive selection, rDNA duplication by TRSs and USCR may be non-adaptive because the duplication of particular rDNA types reduces the variety of rDNA types