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. 2018 Sep 5;11:113. doi: 10.1186/s13045-018-0656-7

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — NEAT1 facilitated CRC cell progression in a DDX5-mediated manner. a CCK-8 and b EdU assay results showing that knockdown of DDX5 partially attenuated the enhanced cell proliferation induced by overexpression of NEAT1 in HCT116-shNEAT1 and SW1116-shNEAT1 cells (*p < 0.05). Representative images of the c Transwell invasion assays and d wound healing assays showing that DDX5 repression rescued the enhanced invasion and migration abilities induced by NEAT1 overexpression (*p < 0.05)