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. 2018 Sep 4;6:e5524. doi: 10.7717/peerj.5524

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©2018 Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, reproduction and adaptation in any medium and for any purpose provided that it is properly attributed. For attribution, the original author(s), title, publication source (PeerJ) and either DOI or URL of the article must be cited.

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Scratch-wound closure was monitored over time. (A) Representative images showed that GDF11 induced significantly decreased migration speed compared with control (GDF11 untreated cells). Black lines in each graph were pointed toward wound edges. (B) Quantification of the remaining wound area uncovered by migrating C17.2 cells revealed a significant inhibition of migration in GDF11-treated cells. The scratch wound areas at time point 0 hour were set to 100%, and the wound areas at other time point were normalized to their respective 0 hours. Bar is 500 µm (n = 5; *p < 0.05).