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A. Addition of sCEA to HNNFb cultures activates a signalling through the AKT-mTOR C1 as well as the STAT3 signalling axes. B. Pharmacological reversal of α-SMA expression in sCEA-activated HNNFbs upon the addition of AKT-mTOR C1 inhibitors palomid-529 or pentoxifylline.
