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. 2018 Sep;33(5):401–408. doi: 10.5001/omj.2018.74

Table 1. Genotype and allele frequency of lipoprotein lipase gene; -93 T<G promoter (rs1800590) polymorphism in study subjects according to body mass index (BMI) and insulin resistance.

Study subjects classified based on BMI p-value*
Obese subjects
n (%)
Non-obese subjects
n (%)
OR (95% CI)
Genotype
TT 257 (83.2) 310 (93.1) Reference Reference
TG 47 (15.2) 22 (6.6) 2.58 (1.51–4.39) < 0.001
GG 5 (1.6) 1 (0.3) 6.03 (0.70–51.95) 0.102
TG+GG 52 (16.8) 23 (6.9) 2.73 (1.63–4.58) < 0.001
Allele
T 561 (90. 8) 642 (96.4) Reference Reference
G 57 (9.2) 24 (3.6) 2.13 (1.55–3.97) < 0.001
Study subjects classified based on insulin resistance status (HOMA)
Insulin resistant subjects n (%) Non-insulin resistant subjects n (%) OR (95% CI) p-value**
Genotype
TT 208 (88.5) 359 (88.2) Reference Reference
TG 24 (10.2) 45 (11.1) 0.92 (0.55–1.56) 0.757
GG 3 (1.3) 3 (0.7) 1.73 (0.35–8.63) 0.506
TG+GG 27 (11.5) 48 (11.8) 0.97 (0.59–1.60) 0.908
Allele
T 440 (93.6) 763 (93.7) Reference Reference
G 30 (6.4) 51 (6.3) 0.98 (0.66–1.48) 0.834

Total number of obese (n = 309) and non-obese subjects (n = 333) (for genotype); total number of chromosomes in obese (n = 618) and non-obese subjects (n = 666) (for alleles); total number of insulin resistant (n = 235) and non-insulin resistant subjects (n = 407) 
(for genotype); total number of chromosomes in insulin resistant (n = 470) and non-insulin resistant subjects (n = 814) (for alleles). 
*adjusted for age and sex.

**adjusted for age, sex, and BMI.

HOMA: homeostasis model assessment; OR: odds ratio; CI: confidence interval.