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. 2018 Feb 22;55(10):7822–7838. doi: 10.1007/s12035-018-0946-7

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© The Author(s) 2018

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 2 — Effect of α-tocopherol treatment on protein carbonyls (PCO) formation used as marker of oxidative stress. PCO content (nmol/mg protein) in the hippocampal homogenates obtained from rats of different experimental groups: αK, treated kainate-exposed; VK, untreated kainate-exposed; αC, treated non-epileptic; VC, untreated non-epileptic. Histograms represent PCO content of three independent measurements in each experimental group (means ± SEM). Statistical analyses performed by one-way ANOVA and Tukey’s post hoc test: **p < 0.01