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. 2018 Aug 24;121(4):342–360. doi: 10.1038/s41437-018-0136-4

Table 2.

Significant genes from the a priori gene list partitioned between components of variance

G ETOH G × ETOH T × ETOH G × ETOH × T 10 20 30
Exons*
Defense
Unique 95(79) 7 2(1) 1 1(1) 7(4) 1(1) 2
Common 21 3 1 0 1 2 1 1
Constitutive 220 14 2 3 6 13 3 6
Ethanol
Unique 29(19) 4(2) 0 1(1) 2 2(2) 2(1) 4(3)
Common 28 5 0 1 0 2 2 2
Constitutive 94 8 4 1 2 6 5 9
Splicing
Unique 13(6) 1 0 0 0 2 2 0
Common 48 2 0 2 0 2 1 3
Constitutive 78 0 2 0 0 1 1 2
Exonic regions
Defense
Common 22 1 0 0 0 0 1 2
Constitutive 77 0 0 0 0 1 0 1
Ethanol
Common 18 0 0 0 1 1 0 0
Constitutive 41 1 1 1 1 0 0 1
Splicing
Common 13 0 0 0 0 1 2 0
Constitutive 42 2 0 1 0 3 0 0
Fragments
Defense
Unique 36(4) 4 2 3 2 0 4 7
Common 69 3 2 1 4 1 5 4
Constitutive 76 2 0 1 0 5 2 2
Ethanol
Unique 9 3 2 1 1 2 1 2
Common 13 7 0 2 3 2 3 2
Constitutive 6 2 0 0 2 0 0 1
Splicing
Unique 25(1) 2 0 0 0 3 0 3
Common 38 4 2 4 2 10 3 5
Constitutive 61 2 0 0 0 2 0 1

Genotype (G), treatment (ETOH), and time (T) are listed separately from tests for differences between ethanol and no ethanol at 10, 20, and 30 min.

*Parentheses indicate the number shared between the “unique” and “consistutive” list as they are not mutually exclusive.