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. 2018 Mar 12;67(7):991–999. doi: 10.1093/cid/ciy213

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© The Author(s) 2018. Published by Oxford University Press for the Infectious Diseases Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — Kaplan–Meier plot comparing in-hospital acute kidney injury (AKI) development in patients with severe and moderately severe malaria treated with either acetaminophen or no acetaminophen (control). Patients were classified as developing AKI if they had a creatinine rise of ≥26.5 µmol/L after admission. A total of 17/62 (27%) patients had AKI during admission: 12/17 (38%) in the control group and 5/12 (16%) in the acetaminophen group. Competing risks regression adjusted by study site was used to assess subdistribution hazard ratio. Patients were censored at the time of creatinine rise meeting Kidney Disease: Improving Global Outcomes criteria and death censored as a competing risk preventing the primary event of interest (AKI) from occurring. Abbreviations: CI, confidence interval; KDIGO, Kidney Disease: Improving Global Outcomes; SHR, subdistribution hazard ratio.