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. 2018 Aug 17;16(4):3345–3352. doi: 10.3892/etm.2018.6615

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Copyright: © Song et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Analysis of IR, IRS-1, PI3K and AKT protein expression by immunohistochemical staining. (A) IR, IRS-1, PI3K and AKT protein expression in the rat livers of each group was detected by immunohistochemical staining (magnification, ×200). (B) Quantification of immunohistochemical staining in each group. *P<0.05 vs. control group; ^#P<0.05 vs. diabetic model group; ^&P<0.05 vs. low-dose sericin group. IR, insulin receptor; IRS-1, IR substrate-1; PI3K, phosphoinositide 3-kinase; AKT, protein kinase B.