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. 2018 Jul 27;25(14):1548–1557. doi: 10.1177/2047487318790722

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© The European Society of Cardiology 2018

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 1. — Relations between adiposity measurements and adipokines. Results are presented as regression coefficients (β) and 95% confidence intervals (CIs) per 1 standard deviation (SD) increase in adiposity measurements. Model II (open squares) is adjusted for age and gender, model III (closed squares) is additionally adjusted for current smoking, lipid-lowering therapy, history of diabetes type 2 and high-sensitivity C-reactive protein (hsCRP). BMI: body mass index; WC: waist circumference; SAT: subcutaneous adipose tissue; VAT: visceral adipose tissue; MCP-1: monocyte chemoattractant protein 1; NGF: nerve growth factor; HGF: hepatic growth factor; PAI-1: plasminogen activator inhibitor 1; MIF: migration inhibitory factor; SAA1: serum amyloid A1.