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. 2018 Sep 19;16(3):1559325818800153. doi: 10.1177/1559325818800153

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© The Author(s) 2018

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — A, Western blot analysis of the expression of NF-κB, COX-2, STAT3, and NrF2 proteins in control, SF-intoxicated, and all treated groups. B, The densitometry analysis of the expression of NF-κB, COX-2, STAT3, and NrF2 proteins in control, SF-intoxicated, and all treated groups. (Data corrected by β-actin and expressed as protein/β-actin). Data are presented as mean ± SEM (N = 6). ⁺⁺⁺ P ≤ .001 versus control and ***P ≤ .001 versus SF-intoxicated group. COX-2 indicates cyclooxygenase-2; NAC, N-acetylcysteine; NF-κB, nuclear factor-κB; NrF2, nuclear factor erythroid 2-related factor 2; SEM, standard error of the mean; SF, sodium fluoride; STAT-3, signal transducer and activator of transcription 3; TMQ, thymoquinone.