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. Author manuscript; available in PMC: 2018 Oct 1.
Published in final edited form as: Neurobiol Aging. 2018 Jun 11;70:70–77. doi: 10.1016/j.neurobiolaging.2018.06.004

Table 1.

Subject demographics and neuropathologic assessment

Case Age in
years
Gender Break
stage
CERAD
plaque
score
Level of AD
pathologic change
CAA

AD with CAA
type 1
    1 79 M VI 3 High Severe
    2 78 F VI 2 High Severe
    3 68 F VI 3 High Severe
    4 101 M V 2 High Severe
    5 73 M V 2 High Severe
    6 75 M VI 3 High Severe
    7 76 M VI 1 Intermediate Moderate
    8 80 M VI 3 High Severe
AD with CAA
type 2
    1 76 M VI 3 High Severe
    2 93 F VI 3 High Moderate
    3 69 F VI 3 High Severe
    4 86 F IV-V 3 Intermediate-high Moderate
    5 82 F VI 3 High Severe
    6 78 M VI 2 High Severe
    7 68 M VI 2 High Severe
    8 64 F VI 1 Intermediate Severe
    9 96 F VI 2 High Moderate
    10 70 M VI 2 High Moderate
AD without
CAA
    1 65 F VI 2–3 High No CAA
    2 115 F IV-V 2 Intermediate-high No CAA
    3 81 F V 3 High No CAA
    4 63 VI, also DLBD 3 High No CAA
    5 61 M VI 3 High No CAA
    6 70 M VI 3 High No CAA
    7 93 M V-VI 1–2 Intermediate-high No CAA
Nondemented
elderly
    1 71 M II 0 Low No CAA
    2 76 F <IV 0 Not No CAA
    3 66 M I 0 Low No CAA
    4 79 F II 0 Not No CAA
    5 64 F <IV 0 Not No CAA
    6 67 M I 0 Not No CAA
    7 75 F III-IV 0 Low No CAA
    8 65 M I-II 0 Not No CAA
    9 75 M I 0 Not No CAA
    10 89 M <IV 0 Low No CAA

Key: AD, Alzheimer disease; CERAD, Consortium to Establish a Registry for AD; CAA, cerebral amyloid angiopathy; DLBD, diffuse Lewy body disease.

HHS Vulnerability Disclosure