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Model for K+ recycling via H+,K+-ATPase in purinergic regulation of ENaC. Apical K+ recycling by H+,K+-ATPase facilitates normal ATP secretion and downregulation of ENaC by the purinergic signaling system during HS intake. HKα1−/− mice have reduced urinary ATP, possibly due to disruption of the electrochemical ATP-K+ efflux-coupled mechanism.
