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. 2018 Aug 29;12:65. doi: 10.3389/fncir.2018.00065

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Copyright © 2018 Wolter, Möhrle, Schmidt, Pfeiffer, Zelle, Eckert, Krämer, Feil, Pilz, Knipper and Rüttiger.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Loss of GC-B results in reduced responses in auditory steady state response input-output (ASSR I–O) function. (A) Mean ± SEM signal-to-noise ratio (SNR) modulation depth response (carrier frequency 11.31 kHz; stimulus level 40 dB SL; modulation frequency 512 Hz) was not significantly different between genotypes. (B) Mean ± SEM SNR of the response to increasing modulation frequencies (carrier frequency 11.31 kHz; stimulus level 40 dB SL; modulation depth 100%) of GC-B WT (black circles), GC-B HET (green circles) and GC-B KO mice (red circles) did not differ between genotypes. (C) Mean ± SEM SNR modulation I–O function of an amplitude-modulated tone (carrier frequency 11.31 kHz; modulation frequency 512 Hz; modulation depth 100%). GC-B KO I–O function was significantly reduced compared to GC-B WT and GC-B HET mice.