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. 2018 Sep 25;4:58. doi: 10.1038/s41421-018-0062-x

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a–d RER and EE values of sham and RYGB mice (a, b) (n = 12). Quantitative results are shown in c, d. e Oxidation ratio of glucose and fat in sham and RYGB rats (n = 12). f, g Mitochondrial fatty acid oxidative activity in the liver and skeletal muscle from sham and RYGB rats (n = 8). h Enriched metabolic pathways in the rat liver regulated by RYGB (n = 3). Values are shown as the mean ± s.e.m. Significance was calculated using two-way ANOVA with Tukey’s post hoc analysis (c, d) or the two-tailed unpaired t-test (e-g). *P < 0.05 and **P < 0.01 compared with the sham group