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. 2018 Sep 14;29(7):617–637. doi: 10.1097/FBP.0000000000000430

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Copyright © 2018 The Author(s). Published by Wolters Kluwer Health, Inc.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal. http://creativecommons.org/licenses/by-nc-nd/4.0/

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Fig. 1 — Long-term, selective deficits in working memory after traumatic brain injury (TBI) (Martens KM, Vonder Haar C, Swan AA, Emery MA, Clayton ER, Peterson TC, Hoane MR, unpublished data). Rats (3 months of age) received a bilateral frontal, focal TBI and were tested up to 18 months postinjury. (a) TBI caused deficits in the reference version of the Morris water maze (P=0.012), which resolved by 9 months postinjury. (b) TBI also caused deficits in a working memory version of the task where the platform was moved to a novel location daily, but these never resolved (P=0.004).