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. 2017 Mar 8;22(3):433. doi: 10.3390/molecules22030433

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© 2017 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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LPS pretreatment results in enhanced phosphorylation of STAT3. Figure shows representative images (A) and quantification (B) of western blots of possible cardiac pathways in LPS-induced delayed preconditioning. Analysis was performed 24 h after in vivo 0.5 mg/kg low-dose lipopolysaccharide (LPS) treatment. Values are expressed as mean ± S.E.M. (n = 7 in each groups). ^# p < 0.05 vs. control, unpaired t-test. p-STAT3: phospho(Tyr705)-STAT3, p-Akt: phospho(Ser473)-Akt, p-Erk1/2: phospho(Thr202/204)-Erk1/2, GAPDH: glyceraldehyde 3-phosphate dehydrogenase.