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. 2018 Sep 25;8:14369. doi: 10.1038/s41598-018-32717-3

Table 4.

Molecular alterations in IM among the Hp, AG, and GC groups.

No. of IM samples Hp group AG group GC group Hp vs AG group GC vs AG group
n = 21 (%) n = 23 (%) n = 35 (%) P P OR (95% CI)
MSI 8 (38.1) 4 (17.4) 5 (14.3) 0.18 1
CIMP 7 (33.3) 3 (13.0) 19 (54.3) 0.16 0.002 7.92 (1.98–31.60)
Estimate SE P Estimate SE P
CDH1 7 (33.3) 2 (8.7) 7 (20.0) 0.33 0.13 0.01 0.11 0.12 0.35
CDKN2A 0 (0) 1 (4.3) 0 (0) −0.03 0.03 0.26 −0.03 0.02 0.20
MLH1 0 (0) 0 (0) 0 (0)
MGMT 0 (0) 0 (0) 3 (8.6) 0.04 0.07 0.56 0.10 0.06 0.11
MINT1 13 (61.9) 18 (78.3) 26 (74.3) −0.15 0.15 0.30 −0.02 0.13 0.85
MINT31 12 (57.1) 10 (43.5) 24 (68.6) 0.19 0.16 0.24 0.27 0.14 0.07
RUNX3 6 (28.6) 5 (21.7) 17 (48.6) 0.05 0.14 0.71 0.23 0.12 0.07
miR-124a-3 21 (100) 19 (86.4)a 31 (88.6) 0.13 0.11 0.24 −0.02 0.09 0.85
miR-34c 13 (61.9) 12 (54.5)b 20 (58.8)c −0.01 0.17 0.95 −0.01 0.15 0.92

a,b and c indicate 19 of 22, 12 of 22, and 20 of 34, respectively.

IM, intestinal metaplasia; SE, standard error; MSI, microsatellite instability; CIMP, CpG island methylator phenotype.