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. 2018 Sep 26;13(9):e0204119. doi: 10.1371/journal.pone.0204119

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Fig 10 — A: Summary of changes in hepatic ω-6 and ω-3 PUFA-derived oxylipins in response to EtOH exposure. B: A working model wherein multiple ω-6-derived oxylipins (to the left) facilitate liver injury via distinct mechanisms. The deleterious effects of these oxylipins outweigh the benefits of anti-inflammatory and pro-resolving bioactive lipid mediators (to the right), and tips the balance towards liver damage. Abbreviations are listed in S4 and S6 Tables.