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. 2018 Sep 27;8:14457. doi: 10.1038/s41598-018-32780-w

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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PON1^−/− results in a reduction in T cell numbers that is not dependent on the thymic microenvironment. (a) Schematic overview of the strategy for bone marrow transplantation. (b) Chimerisms of donor-derived cells in the peripheral blood (PB) of irradiated recipient rats at 2, 4, and 6 weeks after transplantation with either PON1^+/+ or PON1^−/− BM cells (n = 9). (c) The percentage of GFP-derived T cells in the PB of PON1^+/+ or PON1^−/− recipient rats 6-weeks after transplantation (n = 9). (d) Schematic overview of the strategy for bone marrow transplantation. (e) Chimerisms of donor-derived cells in the PB of irradiated recipient rats from 2, 4, and 6 weeks after transplantation with GFP BM cells (n = 5). (f) The percentage of PON1^+/+- or PON1^−/–derived T cells in the PB of GFP recipient rats 6 weeks after transplantation (n = 5). Data represent the mean ± SD. *P < 0.05; **P < 0.01.