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. 2018 Oct 2;9:4028. doi: 10.1038/s41467-018-06492-8

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Pam18 controls the lateral gate promoting anterograde protein transport. Model of how Pam18 might affect release of membrane-targeted proteins into the lipid bilayer. When Pam18 is bound together with other motor components, the lateral gate is closed, which promotes matrix translocation of substrates. IMS, intermembrane space; IM, inner membrane