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. 2018 Sep 27;9:1092. doi: 10.3389/fphar.2018.01092

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Copyright © 2018 Zheng, Lin, Lin, Lin, Thapa, Lin, Lian, Liu, Chen and Li.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Western blotting showed that SH decreased the expression of inflammatory cytokines, increased the activation of AMPK, suppressed the activation of NF-kB P65 on H9C2 post COCl₂-induced hypoxia. Control, n = 6; 0, CoCl₂ + 0 μ/ml SH, n = 6; 10, CoCl₂ + 10 μg/ml SH, n = 6; 20, CoCl₂ + 20 μg/ml SH, n = 6; 30, CoCl₂ + 30 μg/ml, n = 6. ^∗Compared with control, p < 0.05; ^#compared with 0 μg/ml SH post CoCl₂, p < 0.05; ^∧compared with 10 μg/ml SH post CoCl₂, p < 0.05; ^&compared with 20 μg/ml post CoCl₂, p < 0.05.