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. 2018 Oct 3;24:52. doi: 10.1186/s10020-018-0050-5

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© The Author(s) 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — The effects of MALAT1 on proliferation, invasion and migration of colorectal cancer cells through regulating miR-145 a and b cell proliferation of DLD-1 and HT-29, cell proliferation of si-MALAT1 group was the lowest, NC group and si-MALAT1 + miR-145 inhibitor group was comparative and miR-145 inhibitor group was the highest. c colony formation of DLD-1 and HT-29 cells, the number of clone cells was the least in si-MALAT1 group. The miR-145 inhibitor group had the most clone cells. The number of clone cells was comparative in si-MALAT1 + miR-145 inhibitor group and NC group. d invasion assay of DLD-1 and HT-29 cells. e wound healing assay of DLD-1 and HT-29 cells. ^**P < 0.01, compared with NC group