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. Author manuscript; available in PMC: 2018 Nov 8.
Published in final edited form as: Int J Impot Res. 2018 May 8;30(3):108–116. doi: 10.1038/s41443-018-0021-y

Fig. 1.

Fig. 1

Erectile function is decreased in rats with cavernous nerve injury, which is prevented by treatment with a denitrosylating agent NAC. Sham-injured and BCNI-rats were treated with vehicle or NAC (300 mg/kg/day) starting 2 days before BCNI or sham injury and continuing for 2 weeks after the surgery. Electrical stimulation of the cavernous nerve was performed as voltage response (1, 2, and 4 V) at 16 Hz with square-wave duration of 5 msec for 1 min. Representative ICP and MAP responses to 4 V electrical stimulation (A). The stimulus interval is indicated by a solid bar. Erectile response to electrical stimulation of the cavernous nerve is indicated by maximal ICP/MAP (B) and total ICP/MAP (C). Each bar represents the mean ± SEM of 5-8 rats. *P < 0.05 vs. Sham + Vehicle; #P < 0.05 vs. BCNI + Vehicle.