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. 2018 Oct 6;11:59. doi: 10.1186/s13072-018-0230-0

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© The Author(s) 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 4 — CFP1 binding and histone methylation marks in putative enhancers and comparison to TSSs. a Loci sorted by H3K4me1 signal in 6-kb regions surrounding putative enhancers. Upper panels, ERY; lower, EBV. Note that all data sets, other than CFP1, differ by source between ERY and EBV, which explains prominent differences in signal intensities, for example, of H3K27ac and H3K4me1. b Quantitative comparison of signal intensity in TSSs and putative enhancers, above and below median accessibility (as determined by ATAC-seq data), with cell types indicated. c Plots of ChIP signals for erythroid (left) and lymphoid cells (right), in putative enhancers (lower left graphs) and TSSs (upper right graphs)