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. 2018 Jun 6;5(3):290–299. doi: 10.1016/j.gendis.2018.05.004

Figure 1.

Figure 1

The binding affinity of rosiglitazone (Rosi) and CMHX008 (CMHX) on PPARγ. (A) Chemical structure of Rosi and CMHX. (B) TR-FRET competitive binding assay was used to examine the binding affinity of the human PPARγ in response to rosiglitazone or CMHX008 at concentrations ranging from 0.3 to 1000 nM (n = 3).