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. 2018 Oct 9;9:4173. doi: 10.1038/s41467-018-06696-y

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — Hypothetical model integrating components involved in bud break. Low temperature reduces ABA levels and suppresses SVL expression, leading to induction of FT1 expression and GA biosynthesis, which promotes bud break. In the absence of low temperatures, high levels of SVL expression induce NCED3 and RCAR/PYL, thereby maintaining high ABA levels and sensitivity in buds, ensuring that they remain dormant. SVL subsequently induces TCP18/BRC1, suppressing bud break. Low temperatures trigger reduction in SVL expression and its suppressive effects, followed by bud break