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. 2018 Oct 9;9(10):1032. doi: 10.1038/s41419-018-1058-z

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a–b GO and KEGG analysis of significant target genes. c Heatmap of altered genes after NTZ treatment. d The relative expression of ING1 in LN229 cells after 48 h NTZ treatment. The experiments were repeated 3 times independently, and the bars represent SD. The data were normalized with control group (*P < 0.05). e Kaplan–Meier survival analysis of ING1 for all glioma patients (662 glioma tissue samples). f Kaplan–Meier survival analysis of ING1 for LGG glioma patients (510 LGG tissue samples). Low-grade glioma LGG. g Kaplan–Meier survival analysis of ING1 for GBM patients (152 GBM tissue samples)