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. 2018 Oct 9;9:4172. doi: 10.1038/s41467-018-06366-z

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Key molecular pathways altered in metastatic ACC. Network of canonical pathways that are significantly altered in metastatic ACC based on ingenuity pathway analysis (IPA) (a). Pathways that are predominantly altered in metastatic ACC including ERBB4 signaling (b), retinoic acid receptor signaling (c), G-protein-coupled receptor signaling (d), and platelet-derived growth factor receptor signaling (e). The number within each box represents the percentage of gene mutations in these tumors