Table 2.
Comparisons of CT findings between diabetic patients who develop IKLAS with different HbA1c concentrations
| CT features, n (%) | HbA1c concentrations
|
P-value | ||
|---|---|---|---|---|
| ≤7% (n=4) | 7%–9% (n=5) | ≥9% (n=32) | ||
|
| ||||
| Well-defined margin | 3 (75.0) | 3 (60.0) | 18 (56.0) | 0.87 |
| Multiple abscesses | 2 (50.0) | 2 (40.0) | 18 (56.0) | 0.86 |
| A cystic component | 3 (75.0) | 4 (80.0) | 26 (81.3) | 0.99 |
| Multilocular | 3 (75.0) | 4 (80.0) | 21 (65.6) | 0.85 |
| Gas in abscess | 1 (25.0) | 2 (40.0) | 14 (43.8) | 0.86 |
| Hepatic venousthrombophlebitis | 0 | 0 | 15 (46.8) | 0.03a |
| Septic pulmonary embolism | 1 (25.0) | 0 | 10 (31.3) | 0.40 |
| Pleural effusion | 2 (50.0) | 3 (60.0) | 22 (68.8) | 0.73 |
Note:
a
Diabetic patients with IKLAS in the group with poor glycemic control (47%) had a significant probability of developing hepatic venous thrombophlebitis than those patients with suboptimal or good glycemic control (P=0.03).
Abbreviations: CT, computed tomography; IKLAS, invasive Klebsiella pneumoniae liver abscess syndrome.