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. 2018 Oct 12;9:4228. doi: 10.1038/s41467-018-06620-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Putative damaging variants with MAF < 5% in SLC2A9 (hSLC2Ab). Of the 90 variants included in primary gene-based test, 87 were missense, and 3 were splice variants. Of the 87 missense variants, 77 were found in the hSLC2A9b. The ± signs indicate variants selected for mapping onto the SLC2A9 predicted structure. Beta between −1.5 and −1.0 (−); beta < −1.5 (−−); beta between 1.0 and 1.5 (+); beta >1.5 (++), Orange: predicted transmembrane domains. Amino acid position based on NP_001001290 (ENSP00000422209)