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. 2018 Oct 17;9(11):1058. doi: 10.1038/s41419-018-1076-x

Table 2.

Clinical characteristics of TLE patient

Sex (M/F) Age (years) Course (years) History of AEDs Source of organization Pathologic diagnosis
M 18 3 VPA/LTG/LEV LTN NL, G
M 22 10 CBZ/TPM/PB LTN G
F 14 4 LTG/TPM/PB RTN NL, G
M 39 15 VPA/TPM/LEV LTN NL, G
F 47 20 CBZ/LTG/PHT/PB LTN NL, G
F 55 5 PB/OXC/LEV RTN G
F 21 3 OXC/LEV/PB LTN NL, G
M 17 6 VPA/OXC/LEV RTN NL, G
F 9 2 CBZ/PB/LTG LTN G
M 33 10 TPM/VPA/LEV/PB LTN NL, G
M 28 8 PB/VPA/LEV RTN NL, G
M 43 20 CBZ/TPM/LEV/PB RTN NL, G
F 36 17 CBZ/LTG/TMP/PB LTN NL, G
M 25 5 VPA/LEV/CBZ LTN G
F 48 23 CBZ/VPA/LEV RTN NL, G
M 33 13 VPA/LEV/PHT RTN NL, G
M 27 7 PB/PHT/VPA LTN NL, G
F 40 20 CBZ/PB/LTG/LEV LTN NL, G
M 38 8 CBZ/VPA/LEV/PB RTN NL, G
M 67 33 PHT/TPM/PB LTN G

E epilepsy, M male, F female, AEDs antiepileptic drugs, VPA valproic acid, LTG lamotrigine, LEV levetiracetam, CBZ carbamazepine, TPM topiramate, PB phenobarbital, PHT phenytoin, OXC oxcarbazepine, LTN left temporal neocortex, RTN right temporal neocortex, NL neuronal necrosis, G glios